超长技术类文章，很多生僻词汇如何记笔记？

Pope_Xie

如题，楼主在碰到下面这样的文章时，就会自动脱线，觉得很多名词，都有逻辑关系，譬如A削弱B，C削弱D，正式考试环境下时间紧张，记笔记时会觉得很抓快。请各路大侠，赐教对于这类文章记笔记的方法。

Caffeine, the stimulant in coffee, has been called
“the most widely used psychoactive substance on
Earth.” Snyder, Daly, and Bruns have recently
proposed that caffeine affects behavior by
countering the activity in the human brain of a
naturally occurring chemical called adenosine.
Adenosine normally depresses neuron firing in many
areas of the brain. It apparently does this by
inhibiting the release of neurotransmitters,
chemicals that carry nerve impulses from one
neuron to the next.
Like many other agents that affect neuron
firing, adenosine must first bind to specific
receptors on neuronal membranes. There are at
least two classes of these receptors, which have
been designated A1and A2
. Snyder et al. propose
that caffeine, which is structurally similar to
adenosine, is able to bind to both types of
receptors, which prevents adenosine from attaching
there and allows the neurons to fire more readily
than they otherwise would.
For many years, caffeine’s effects have been
attributed to its inhibition of the production of
phosphodiesterase, an enzyme that breaks down
the chemical called cyclic AMP. A number of
neurotransmitters exert their effects by first
increasing cyclic AMP concentrations in target
neurons. Therefore, prolonged periods at the
elevated concentrations, as might be brought about
by a phosphodiesterase inhibitor, could lead to a
greater amount of neuron firing and, consequently,
to behavioral stimulation. But Snyder et al. point out
that the caffeine concentrations needed to inhibit
the production of phosphodiesterase in the brain
are much higher than those that produce
stimulation. Moreover, other compounds that block
phosphodiesterase’s activity are not stimulants.
To buttress their case that caffeine acts
instead by preventing adenosine binding, Snyder et
al. compared the stimulatory effects of a series of
caffeine derivatives with their ability to dislodge
adenosine from its receptors in the brains of mice.
“In general,” they reported, “the ability of the
compounds to compete at the receptors correlates
with their ability to stimulate locomotion in the
mouse; i.e., the higher their capacity to bind at the
receptors, the higher their ability to stimulate
locomotion.” Theophylline, a close structural relative
of caffeine and the major stimulant in tea, was one
of the most effective compounds in both regards.
There were some apparent exceptions to the
general correlation observed between adenosinereceptor binding and stimulation. One of these was
a compound called 3-isobutyl-1-methylxanthine
(IBMX), which bound very well but actually
depressed mouse locomotion. Snyder et al. suggest
that this is not a major stumbling block to their
hypothesis. The problem is that the compound has
mixed effects in the brain, a not unusual occurrence
with psychoactive drugs. Even caffeine, which is
generally known only for its stimulatory effects,
displays this property, depressing mouse
locomotion at very low concentrations and
stimulating it at higher ones

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