2011年1月GMAT阅读机经(至1.31)(三十一)。 2.1.8 肌肉疲劳★△ V1(770) 关于肌肉疲劳, 说是一开始科学家认为是产生一种lattice acid导致, 下一段讲好像不是这样, 因为加拿大的研究人员发现在海拔高的地方运动员感觉疲劳, 但那个acid并不多。 然后好像是南非的生理学家又发现了一个什么。 最后一段就提出来好像是大脑的什么信号,其他机制让人疲劳, 而且这个解释可以解释老的理论和新的发现其实并不矛盾云云。 V2(740) 还有个讲肌肉fatigue的。说190×还是哪年研究出来肌肉一种酸的含量到一定程度会引发肌肉fatigue. 但是198×年研究人员发现blablabla... 第三段得出结论说,肌肉fatigue 不一定是acid的原因,有可能是大脑控制各种resource发现你要不行了 然后就给你个信号说你不行了,但是其实你还是行的。fatigue很可能只是主观因素造成的 V3(720) 2. 肌肉劳累跟acid 神马到底啥关系那篇。以前寂静考古的非常正确。我记得月度寂静附录了一篇全英的文章吧,跟那个几乎一模一样。参考那个没错的。 P1:什么有个诺贝尔奖得主提出神马观点, P2:做了个试验 P3:总结这个实验的内容说明了什么。 V4第三篇是那个muscle fatigue.寂静里头基本上也讲得挺清楚的了,记得有一题是问第二段作者提cannada那个研究有什么作用之类~~~我忘了选什么了~~ V5(V39) 4.肌肉疲劳什么的。一开始一个noble prize的人说the function of muscle will brake when muscle detects high level of lactic acid。但是后来有几个Canadian scientist做了个实验发现the muscle acts so even when the level of lactic acid is low,然后又有个什么实验(不记得了)。最后一段说其实第一段的那个theory不是完全的错,但是真正决定什么时候stop the muscle function的是neural system。所以这个过程是subjective而不是Objective(有道题问了这个)。 V6(V33)第三篇是肌肉疲劳,一定好好看highlight的类似物,几乎就是原文重现。 考古人的肌肉(KG)(【类似原文】)V1: 考了一篇第一段说传统观点说人的肌肉劳累是因为肌肉的什么酸acid增加了 第二段说但是现在加拿大的一票科学家说运动员劳累的时候这个酸没有增加啊,还有一个地方的科学家(好象是北非的,很神奇)也举出了反例. 最后一段说什么传统的观点说的酸增加确实对了但是地方不对.然后用计算机证明再解释了一通. 问题考了一个是最后一段的作用,这是我的最后一篇了没大有时间看了. V2:(710) Theory 1: muscles go into automated brake when lactic acid builds up after exhausting exercise, leading to fatigue. Theory Two: psychologists argue central nervous system controls the sense of extreme fatigue to keep body from collapse; psychologists say that theory 1 is right about lactic build up, however wrong in the "location".( location=Central nervous system) V3:(V 40) 第一段:1922年有个诺贝尔的理论,人劳累,是因为肌肉释放的酸达到极限,从而让肌肉休息。 第二段:加拿大的科学家有异议。而南非的科学家取出反例,在一种特定的情况下(这种情况可以导致肌肉释放的酸不会大幅增加),对运动员研究发现他们疲劳的时候酸的含量很低,30%的肌肉已经休息了。虽然这些运动员说他们已经很累,达到极限了。 第三段:科学家们就提出假设来解决这个问题,指出1922年有个诺贝尔的理论是部分正确的。但是人的劳累其实不是客观事实,而是人的主观情绪。然后这个理论还能够解释部分现象。 类似原文:By pipilovelail
Interestingly—or unnervingly, depending on how you look at it— someresearchers are uncovering evidence that Stanovnik’s rule of thumb might beright. A spate of recent studies has contributed to growing support for thenotion that the origins and controls of fatigue lie partly, if not mostly, within the brain and the central nervoussystem. The new research puts fresh weight to the hoary coaching cliché: you only think you’re tired.From the time ofHippocrates, the limits of human exertion were thought toreside in the muscles themselves, ahypothesis that was established in 1922 with the Nobel Prize-winning work ofDr. A.V. Hill. The theory went like this: working muscles, pushed to their limit, accumulated lactic acid.When concentrations oflactic acid reached a certain level, so theargument went, the muscles could no longer function. Musclescontained an ‘‘automatic brake,’’ Hillwrote,‘‘carefully adjusted by nature.’’ Researchers, however, have long noted a link between neurologicaldisorders and athletic potential. In the late 1800’s, the pioneering French doctor Philippe Tissiéobserved that phobias and epilepsy could be beneficial for athletic training. Afew decades later, the German surgeon August Bier measured thespontaneous long jump of a mentally disturbed patient, noting that it compared favorably to theexisting world record. These types of exertions seemed to defy the notion ofbuilt-in muscular limits and, Biernoted, were made possible by‘‘powerful mental stimuliand the simultaneous elimination of inhibitions.’’ Questionsabout the muscle-centered model came up again in 1989 when Canadian researcherspublished the results of an experiment called Operation Everest II, in which athletes did heavy exercise inaltitude chambers. The athletes reached exhaustion despite the fact that theirlactic-acid concentrations remained comfortably low. Fatigue, it seemed, might be caused by something else.In 1999, three physiologists from the University ofCape Town Medical School in South Africa took the next step. They worked agroup of cyclists to exhaustion during a 62-mile laboratory ride and measured, via electrodes, the percentage of leg muscles they were usingat the fatigue limit. If standard theories were true, they reasoned, the body should recruit more muscle fibers asit approached exhaustion — a natural compensation for tired, weakening muscles.Instead, the researchers observed the opposite result.As the riders approached complete fatigue, the percentage of active muscle fibersdecreased, until they were using only about 30 percent.Even as the athletes felt they were giving their all, the reality was that more of their muscleswere at rest.Was the brain purposely holding back the body?‘‘It was as if thebrain was playing a trick on the body, to save it,’’says Timothy Noakes, head of the Cape Town group. ‘‘Which makes alot of sense, if you think about it. In fatigue, it only feels like we’re going to die. Theactual physiological risks that fatigue represents are essentiallytrivial.’’From this, Noakes and his colleagues concluded that A.V.Hill had been right about the automatic brake, but wrong about its location. 这个以下似乎就不一样了。 Theypostulated the existence of what they called a central governor:a neural systemthat monitors carbohydrate stores, thelevels of glucose and oxygen in the blood, the rates of heat gain and loss, and work rates. The governor’s job is to holdour bodies safely back from the brink of collapse by creating painfulsensations that we interpret as unendurable muscle fatigue.Fatigue, the researchers argue, is less an objective event than a subjectiveemotion —the brain’s clever,self-interested attempt to scare you into stopping.The way past fatigue, then, is to return the favor: to fool the brain by lying to it, distracting it or even provoking it. (Thatsaid, mental gamesmanship can never overcome a basiclack of fitness. As Noakes says, thebody always holds veto power.)‘‘Athletes and coaches already do a lot of thisinstinctively,’’Noakes says.‘‘What is a coach, after all, but a technique for overcoming thegovernor?’’The governor theory is far from conclusive, but some scientists are focusing on awalnut-size area in the front portion of the brain called the anteriorcingulate cortex. This has been linked to a host of core functions, including handling pain, creating emotion and playing a key role inwhat’s known loosely as willpower. Sir Francis Crick, the co-discoverer of DNA, thought the anterior cingulate cortex to bethe seat of the soul. In the sports world, perhaps no soul relies on it more than JureRobic’s.Some people‘‘have the ability to reprocess the pain signal,’’ says Daniel Galper, a senior researcher in the psychiatrydepartment at the University of Texas Southwestern Medical Center at Dallas.‘‘It’s not that they don’t feel the pain; they just shift their brain dynamicsand alter their perception of reality so the pain matters less. It’s basicallya purposeful hallucination.’’Noakes and his colleagues speculate that thecentral governor theory holds the potential to explain not just feats ofstamina but also their opposite:chronic fatigue syndrome (a malfunctioning, overactive governor, in this view).Moreover, the governor theory makes evolutionary sense.Animals whose brains safeguarded an emergency stash of physical reserves mightwell have survived at a higher rate than animals that could drain their fueltanks at will.The theory would also seem to explain a sports landscape in whichultra-endurance events have gone from being considered medically hazardous tosomething perilously close to routine. The Ironman triathlon in Hawaii —a2.4-mile swim, 112-mile bike ride and marathon-length run—wasthe ne plus ultra in endurance in the 1980’s, but has now been topped by the Ultraman, which is more than twice as long. Once obscure, the genre known as adventure racing, which includes 500-plus-mile wilderness raceslike Primal Quest, has grown to more than 400 events each year.Ultramarathoners, defined as those who participate in runningevents exceeding the official marathon distance of 26.2 miles, now number some 15,000 in the United States alone. The underlyingphysics have not changed, butrather our sense of possibility. Athletic culture, like Robic, has discovered a way to tweak its collectivegovernor. 考古: 版本1 p1, 老观点,肌肉运动后会产大量acid,新观点,不是这样的p2, 新的实验发现cylist运动了一段时间,没有增加acid?,反而减少了,与老观点不符p3,另一个例子,记不清了,然后结论是运动是由大脑主观调节的。